Bibliografía
[1]
Rahman, R., Gopinath, D., Buajeeb, W., Poomsawat, S., & Johnson, N. W. (2022). Potential Role of Epstein–Barr Virus in Oral Potentially Malignant Disorders and Oral Squamous Cell Carcinoma: A Scoping Review. Viruses, 14(4), 801. https://doi.org/10.3390/v14040801
[2]
Elkhalifa, A. M. E., Nabi, S. U., Shah, O. S., Bashir, S. M., Muzaffer, U., Ali, S. I., Wani, I. A., Alzerwi, N. A. N., Elderdery, A. Y., Alanazi, A., Alenazy, F. O., & Alharbi, A. H. A. (2023b). Insight into Oncogenic Viral Pathways as Drivers of Viral Cancers: Implication for Effective Therapy. Current Oncology, 30(2), 1924-1944. https://doi.org/10.3390/curroncol30020150
[3]
Sanjuan, N. (s. f.). ONCOGÉNESIS VIRAL. Facultad de Medicina (UBA). https://www.fmed.uba.ar/sites/default/files/2020-03/RESUMEN%20DE%20ONCOG%C3%89NESIS%20VIRAL.pdf
[4]
Isabel Toro-Montoya, A. (2023). Virus de Epstein-Barr: más que una mononucleosis infecciosa. BvSalud. https://docs.bvsalud.org/biblioref/2023/02/1414241/51-64-veb.pdf
[5]
Hau, P. M., & Tsao, S. W. (2017). Epstein-Barr Virus Hijacks DNA Damage Response Transducers to Orchestrate Its Life Cycle. Viruses, 9(11), 341. https://doi.org/10.3390/v9110341
[6]
Li, J. S. Z., Abbasi, A., Kim, D. H., Lippman, S. M., Alexandrov, L. B., & Cleveland, D. W. (2023). Chromosomal fragile site breakage by EBV-encoded EBNA1 at clustered repeats. Nature, 616(7957), 504–509. https://doi.org/10.1038/s41586-023-05923-x
[7]
Stewart, J. A., & Damania, B. (2023). Human DNA tumor viruses evade uracil-mediated antiviral immunity. PLoS pathogens, 19(3), e1011252. https://doi.org/10.1371/journal.ppat.1011252
[8]
Zhang, Y., Guo, W., Zhan, Z., & Bai, O. (2024). Carcinogenic mechanisms of virus-associated lymphoma. Frontiers In Immunology, 15. https://doi.org/10.3389/fimmu.2024.1361009
[9]
Patra, T., Meyer, K., Ray, R. B., & Ray, R. (2021, 1 marzo). Hepatitis C virus mediated inhibition of miR-181c activates ATM signaling and promotes hepatocyte growth. PubMed. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7008082/
[10]
Machida, K., Cheng, K. T., Sung, V. M., Shimodaira, S., Lindsay, K. L., Levine, A. M., Lai, M., & Lai, M. M. C. (2004). Hepatitis C virus induces a mutator phenotype: Enhanced mutations of immunoglobulin and protooncogenes. Proceedings Of The National Academy Of Sciences Of The United States Of America, 101(12), 4262-4267. https://doi.org/10.1073/pnas.0303971101
[11]
Vescovo, T., Refolo, G., Vitagliano, G., Fimia, G., & Piacentini, M. (2016). Molecular mechanisms of hepatitis C virus–induced hepatocellular carcinoma. Clinical Microbiology And Infection, 22(10), 853-861. https://doi.org/10.1016/j.cmi.2016.07.019